I. Progress in Amyloidosis Research.- A Brief History of International Symposia on Amyloidosis.- Statistical Analysis of Amyloid Activities Since 1950 -- Basis for an Autonomous Journal.- II. Amyloidogenesis.- Anti-Casein Antibodies in the Serum of Patients with Amyloidosis.- Experimental Systemic Amyloidosis in Mice-Induction of Amyloidosis by Immunization with Syngeneic Organ Extracts and Immunological Investigation.- Secondary Amyloidosis Associated with Chronic Arthritis in Rabbits.- Cloning and Sequence Analysis of Cdnas Encoding Mouse C-Reactive Protein.- Systemic Amyloidosis: Tissue Specific Variability in the Primary Structure of Amyloid Proteins.- Fourier Transform Infrared Spectroscopy of Air-Dried and Heavy Water Suspended AA and AL Amyloid Fibril Preparations of Different Species.- Replacement of SAA From the HDL-SAA Complex by APO AI and APO All: HDL Has Higher Binding Capacity for SAA than for AA.- Aposaa and AA: Precursor and Product Revisited.- SAA2 Deposition in the Spleen of the Mouse.- In Vitro Formation of Amyloid Fibrils from Murine Serum Amyloid A Protein (SAA).- Induced AA Amyloid in Hamster: On the Amyloid Enhancing Factor and Protein AA-Cross Reacting Components of Intermediate.- Deposition of Amyloid a Fibrils in Spleen is Accompanied by Decreased Hepatic and Splenic and Increased Macrophage Serum Amyloid a Expression.- Immunolocalization of Heparan Sulfate Proteoglycans to AA Amyloid Deposition Sites in Spleen and Liver During Experimental Amyloidosis.- Renal Peritubular Amyloid Deposition in the Experimental Murine Amyloidosis.- Ultrastructural Study of the Spleen in Experimental Murine Amyloidosis Using the Protein A-Gold Immunocytochemical Technique.- Experimental Amyloidosis of Rabbit Induced by Saponin.- Ultrastructural Investigation of Spontaneously Occurring Amyloidosis in White Pekin Ducks.- Enhanced Degradation of Serum Amyloid Protein SAA in vivo by Proteolytic Enzymes.- Degradation Studies on Plasma Prealbumin.- Protease and Protease Inhibitors in Familial Amyloidotic Polyneuropathy.- A Study of Dipeptidyl Aminopeptidase IV in the Sera of Japanese FAP Patients.- III. Al Amyloidosis.- Primary Structure of A V K II Light Chain from a Patient with Amyloid Arthropathy.- Molecular Heterogeneity of Amyloid Fibril Proteins in Primary Amyloidosis.- The Amino Acid Sequence of a Carbohydrate-Containing Immuno-Globulin-Lght-Chain-Type Amyloid Fibril Protein (AL).- Structural Analysis of a Lambda IV Primary Amyloid Protein.- Al Amyloidosis (MIY) with Intestinal Tract Involvements and the Chemical Analysis of its BJP (MIY, NIG-84).- Lambda Predominance of Bence Jones Protein (BJP) in Al Amyloidosis Among Japanese Patients.- Immunohistochemical Typing of Tissues From 100 Patients with Systemic Amyloidosis.- Expression of Plasma Cell-Associated Non-Lght Chain Antigens in Patients with Plasma Cell Dyscrasia and Amyloidosis.- Use of Various Electroimmune Techniques in the Diagnosis and Follow-Up of Patients with Al Amyloidosis.- IV. AA Amyloidosis.- The Structure of Protein AA and its Correlation to the Tissue Distribution of Amyloid.- Amino Acid Sequence Composition of AA Amyloid Fibrils in Cystic Fibrosis.- Phylogenetic Analysis of Amyloid a Protein.- Characterization of Serum Amyloid a (SAA) Protein in the Shpep.- Structural Studies of SAA and AA from Horse and Cow.- SAA Versus CRP Serum Levels in Different Inflammatory Conditions, Studied by Elisa Using Polyclonal Anti-AA and Monoclonal Anti-SAA Antibodies.- Monoclonal Antibody to Saal1-3 Isomer Better Discriminant of the Acute Phase Reaction to Cancer and Monitor of Disease Progression than the Polyclonal Antibody to Human AA.- Monoclonal Antibody Against Synthesized Short Peptides Corresponding to Human Amyloid a Protein.- Diagnosis of AA-Amyloidosis Using Monoclonal Antibodies: Application to Paraffin and Ultrathin Sections as Well as to Urinary Sediments.- Influence of Serum Amyloid a on Human Lecithin: Cholesterol Acyltransferase Activity.- Structure and Variation in the Human Serum Amyloid a Gene Family.- Serum Amyloid a: Characterization of Three Cdnas in One Individual and Complete Structure of Their Corresponding Protein Products.- Regulation of Human SAA Gene Expression.- Localization of Amyloid SAA Gene Expression in Mouse Liver by in Situ Hybridization.- Rat Liver and Lung Express Serum Amyloid a Related mRNAs.- Identification of Cells Expressing SAA3 Mrna by in Situ Hybridization.- Diverse Gene Structure for Serum Amyloid a Protein in Wild Mice.- V. Familial Amyloid Polyneuropathy (FAP): Chemical Study.- Two new DNA-Based Tests for Mutations Causing Familial Amyloidotic Polyneuropathy.- Epitope Mapping of TTR (Prealbumin) and TTR (Met30) with Monoclonal Antibodies.- Amyloid Fibril Formation by Polymerization of Abnormal Transthyretin.- Demonstration of Transthyretin (Prealbumin) Variants in Italian Kindreds with Familial Amyloidotic Polyneuropathy.- Abnormal Transthyretin in Familial Amyloidotic Polyneuropathy in Japan and Sweden.- Elderly Asymptomatic Carriers and Late-Onset Cases of Familial Amyloidotic Polyneuropathy Detected by Radioimmunoassay.- Studies on the Fate of Normal and Variant Prealbumin in the Circulation.- Studies on Transthyretin Metabolism in the Nervous System.- Alpha-Adrenergic Receptors in Familial Amyloidotic Polyneuropathy.- DNA Tests for Four Prealbumin Mutations in Hereditary Amyloidosis.- Prealbumin Gene Expression in Control Subjects and Individuals with Familial Amyloidotic Polyneuropathy.- Diagnosis of Familial Amyloid Polyneuropathy by Recombinant DNA Techniques in Relation with Clinical Features.- Haplotype Analisis of Familial Amyloidotic Polyneuropathy: an Evidence for Multiple Origins of the Val?Met Mutation.- DNA Polymorphisms Analysis in Families with Familial Amyloidotic Polyneuropathy (FAP) From Different Ethnic Origins.- RFLP Analysis of Mutated Transtyyrejin in Vitreous Amyloidosis.- Familial Amyloidotic Polyneuropathy in Sweden -- RFLP Analysis in Patients and in Healthy Controls.- A Potential Animal Model for Familial Amyloidotic Polyneuropathy Through Introduction of Human Mutant Transthyrein Gene Into Mice.- Toward Establishment of an Animal Model for Familial Amyloidotic Polyneuropathy: Generation Characterization of Transgenic Mice.- Localization of Prealbumin mRNA in Fetal and Adult Mice.- VI. Familial Amyloid Polyneuropathy (FAP): Clinical and Morphological Study.- Is the Homozygofs State for Hereditary Amyloid Neuropathy (HAN) -- Type I (Portuguese Andrade) Incompatible with Early Intrauterine Life'.- Familial Amyloid Polyneuropathy: A Clinical Scale for the Evaluation of the Peripheral Neuropathy.- Familial Amyloidotic Polyneuropathy: Screening of the Population at Risk in Portugal.- Familial Amyloid Polyneuropathy (Portuguese Type): Study of Asymptomatic Carriers.- Late-Onset Forms of Familial Amyloid Polyneuropathy (Portuguese Type): a Reappraisal.- Genetic Study of Late-Onset in Hereditary Amyloid Neuropathy (HAN) -- Type I (Portuguese Andrade).- Genetic Expression of a Variant Prealbumin in Typical and Late-Onset Japanese Family with Familial Amyloidotic Polyneuropathy.- Natural History of the ECG Abnormalitis in FAP Portuguese Type: a Follow-Up Study with ECG Holter Monitoring and Intracardiac Electrophysiological Study.- Development of High-Grade Ventricular Arrhythmias in Familial Amyloid Polyneuropathy.- Early Detection of Amyloid Heart Disease by Technetium-99M-Pyrophosphate Single-Photon Emission-Computed Tomography: a Study with Familial Amyloid Polyneuropathy.- Impaired Left Ventricular Diastolic Properties in Patients with Familial Amyloid Polyneuropathy: A Study by Computerized M-Mode Echocardiography.- Moninvasive Evaluation of Left Ventricular Systolic and Diastolic Time Intervals in Familial Amyloid Polyneuropathy.- Molecular Analysis of a Variant Type of Familial Amyloidotic Polyneuropathy (FAP) Showing Cerebellar Ataxia and Pyramidal Tract Signs.- Morphometric Study on Changes in Sural Nerves and Comparison with Clinical Manifestations in Familial Amyloidosis.- Sural Nerve Biopsy in Familial Amyloidotic Polyneuropathy: a Morphological and Morphometric Polyneuropathy.- Clinicopathological Studies on Nephropathy of Familial Amyloid Polyneuropathy in Japan.- Pathology of Familial Amyloidotic Polyneuropathy Occurring in Kumamoto.- Pathological Studies on Familial Amyloidosis.- VII. Cerebral Amyloid.- A Unique Protein That Shares a Common Epitope with Senile Plaque in Alzheimer''s Disease Brain.- Preparation of a Monoclonal Antibody Against a Synthetic Brain Amyloid Beta Peptide (Nl-10) and Distribution of an Immunoreactivity in Serum.- Biochemical and Molecular Studies of Native and Synthetic ß-Amyloid Protein in Alzheimer''s Disease.- ß-Amyloid CDNA Cloned from Alzheimer''s Disease Brain.- ?-1-Antichymotrypsin a Serine Protease Inhibitor is a Component of the Amyloid Deposits in Alzheimer''s Disease.- Immunohistochemical Study of Alzheimer Disease Using Antibodies to Synthetic Amyloid and Fibronectin.- Monoclonal Antibody AM34 Detects New Proteins Associated with Secondary Amyloidosis.- Corpora Amylacea in Aging and Alzheimer''s Brain: Immunolocalization of Chondroitin Sulfate and Heparan Sulfate Proteoglycans.- Ubiquitin: A Newly Identified Component of Paired Helical Filaments.- Ultrastructure of Amyloid Fibrils in Alzheimer''s Disease and Down''s Syndrome.- An Isolate of Families with Hereditary Cystatin C Amyloid Angiopathy and Cerebral Haemorrhage in the South of Iceland.- Distribution of Cystatin C Amyloid Deposits in the Icelandic Patients with Hereditary Cystatin C Amyloid Angiopathy.- Studies on the Cystatin C Gene in Patients with Hereditary Cystatin C Amyloid Angiopathy (HCCAA) with Cerebral Haemorrhage.- Possibly "Familial" Cerebral Amyloid Angiopathy in Japan: Immunohistochemical Identification of Gamma-Trace.- VIII. Other Types of Amyloid.- Beta-2 Microglobulin Amyloidosis.- Chemical Analysis of Beta2-Microglobulin Derived Amyloid In Patient
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